I was gonna try and give a mechanistic response to the whole cholesterol talk, but I'll boil it down to this: although you do synthesize most of your own cholesterol, 50% of your dietary cholesterol will wind up, over the course of 8-48 hours, in circulation. If you have a consistently high dietary cholesterol intake, it will bump up your LDL which would lead to pathology. However, two eggs only is about 1/5th of your dietary *loss* per day of cholesterol (192 mg abs, vs 1 g lost in the stool). Although not the most effective way to battle high cholesterol, lowering your own intake doesn't hurt.
There are a whole host of factors to consider, ranging from control of absorption, control of synthesis, and control of LDL vs HDL levels before a reasoned answer can be given. Practically speaking though, the most effective pharmaceutical therapy relies on directly targeting cholesterol synthesis and elimination, not dietary uptake.
I disagree with this statement though:
"dietary cholesterol doesn't affect blood cholesterol in any permanent way"
unless there are some facts behind that. Cholesterol enters through intestinal tissue, winds up in the liver, then is processed for delivery, along with synthesized cholesterol. Past the liver there is no difference between what you ate and what you made, so I don't see why if you had a consistently high cholesterol diet it would somehow not influence blood cholesterol.